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Then researchers proposed that heterozygous knockout mice appeared to be a good animal model for individuals homozygous for the C667T polymorphism, whereas the homozygous null mice were a better one for severely MTHFR-deficient individuals 19

Hirata, H

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These include regulation of the systemic metabolic control [23], fine-tuning of the endothelial function via nitric oxide (NO) scavenging and NO bioavailability adjustment [24, 25], modulation of innate immunity (acting as immune sentinels) [26], regulation of cytokine and chemokine levels in circulation [27], and production of ROS via hemoglobin- or heme-mediated pathways (as a part of the immune response to pathogens) [28]

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