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The mitochondrial morphological damage and reduction in cristae induced by ferroptosis inducers were significantly ameliorated by PPARa over-expression, while this protective effect was inhibited by icFSP1 administration or PPARa knockdown (Fig S2a)

doi:10.1146/annurev.pharmtox.45.120403.095857 [PubMed] 15

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Significant heritability, or familial influence, has also been reported for several aspects of neurologic-related responding (Sanders et al., 2006), which suggest that neurological deficits may represent a biological marker of SZ risk

Mitochondrial mechanisms in Treg cell regulation: Implications for immunotherapy and disease treatment

J., Lin, H