The antioxidant effects result from the (in)direct capacity to deactivate reactive oxygen species (ROS), such as O 2 - and H 2 O 2 , either by riboflavin in its reduced form, as dihydroriboflavin, or as a cofactor of antioxidative enzymes, such as glutathione peroxidase, superoxide dismutase, glutathione reductase, and catalase (as reviewed in refs
10.1021/acsnano.0c03080 Summary Keywords glutathione exhaustion, reduction-response, metal nanomaterials, TME regulation, cancer therapy Citation Liu P, Hao L, Liu M and Hu S (2023) Glutathione-responsive and -exhausting metal nanomedicines for robust synergistic cancer therapy
Exogenous sources also contribute significantly to free radical formation [3]
Certain medications can affect vitamin B12 absorption or metabolism, potentially altering the efficacy of the methylcobalamin component
A fibrolytic potential in the human ileum mucosal microbiota revealed by functional metagenomic
The inclusion criteria for this study were as follows: (1) aged 2079 years with a body mass index (BMI) between 18.5 and 28 kg/m 2 (BMI 28 kg/m 2 was classified as obese)
