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Nonetheless, there is still some controversy on the in vivo involvement of FFAR2 and FFAR3 in GLP-1 modulation (301, 302), with some reports indicating that blockade of GPR43 in vitro releases GLP-1 (303) and others indicating different mechanisms of action, with FFAR2 releasing PYY from intestinal L-cells (81), while FFAR3 restricted to submucosal neuronal activity (295) despite its apparent expression by the majority of enteroendocrine cells (83)

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Supplemental alpha-lipoic acid is usually provided at much higher amounts than food sources

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doi:10.1089/ARS.2009.2695 Morris D, Khurasany M, Nguyen T, et al