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Despite the differences among theories, current research agrees that epigenetic alterations and hormonal dysregulation, leading to inflammation, are central features of endometriosis (12), with reactive oxygen species (ROS) serving a key role in this inflammatory response (4)

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However, in the blood of ME/CFS patients, excessive oxidative stress response to exercise can be measured via different plasma markers (192), and in mitochondria from ME/CFS patients, defects have been detected in CoQ10 and in Complex V (aka ATP synthase) (182, 190)

mTORC2 in the center of cancer metabolic reprogramming

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