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The senescent levels of PDL3 and PDL9 chondrocytes were assessed by senescence-associated beta-galactosidase (SA--gal) staining and qRT-PCR analysis of senescence-associated marker genes

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In contrast, total BA content in Insr P1195L/ +/HFD liver was not different from that in WT/HFD liver, suggesting that BA content might be maintained through various compensatory mechanisms, such as altered BA synthesis, absorption and secretion