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Other variants, such as those in CARD9 , also play a role in UC susceptibility

In addition to this attack, which might be caused by homocysteinylation of the vascular wall proteins by homocysteine and/or its oxidized products (homocysteic acid, homocysteine sulfinic acid, and homocysteine thiolactone), in vitro and in vivo work suggests that reduced homocysteine can inhibit endothelial cell turn-over (Wang et al, 1997) and promote smooth muscle cell proliferation (Lubec et al, 1996b

The studies summarized below are presented with these contextual limitations made explicit

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doi: 10.1016/j.schres.2021.03.018 36 DiSabatoD.QuanN.GodboutJ