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However, when researchers treated these damaged immune cells with ROS-lowering agents like NAC in the laboratory setting, the intervention successfully reduced the intracellular oxidative damage and reversed the functional cellular defects

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showed that exposure to nickel chloride in vitro induced lytic cell death and eryptosis, which is mediated by Ca 2+ influx and p38 MAPK signaling

It is an essential component of various enzyme systems that neutralize free radicals

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