In contrast, mutant TP53 has been proved to drive the glycolysis by activating the RhoA/ROCK/GLUT1 signaling cascade (164), repress the catabolic activities, such as fatty acid oxidation (FAO), by inhibiting 5-AMP-activated protein kinase (AMPK) pathways, and enhance the anabolic processes, such as enhanced fatty acid synthesis (174)
[7] A normal fasting blood sugar level is less than 100 milligrams per deciliter (mg/dl)
Modelling Microglia Activation and Inflammation-Based Neuroprotectant Strategies During Ischemic Stroke
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