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Inherited abnormalities of 5-oxoprolinase (Figure 2, reaction 3), which are autosomal recessive traits, cause the accumulation of this enzymes substrate (5-oxoproline) and generate 5-oxoprolinemia and 5-oxoprolinuria

We were first interested in monitoring the response of NAPstar3b to exogenous H 2 O 2 in cells subjected to glucose starvation, comparing them to cells continuously exposed to 10 mM glucose

The 3.48% reduction in apoptotic cells did not reach statistical significance, though the molecular markers (caspase-3, PARP, Bcl-2/Bax) were all significant

Brock, Ph.D

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