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Preclinical work has suggested that glucagon receptor activation may contribute to changes in energy expenditure and substrate utilization, while GLP-1R and GIPR signaling influence intake-related and endocrine regulatory pathways

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The magnitude and severity of age-associated mitochondrial impairment in these organs becomes clear via the molecular data which shows that old mice had abnormalities in mitochondrial biogenesis and energy metabolism (PGC1), and impaired mitochondrial -oxidation of fatty-acids (PPAR), ATP synthesis (ATP5A), and mitophagy (PINK1)

High throughput screening of millions of compounds across diverse chemical space is a starting point