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SLC25A39 and its paralogue SLC25A40 were found to be essential for mitochondrial import of glutathione, providing protection against oxidative stress and mitochondrial dysfunction (6,7)

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(2019) 30:46276.e6

There is ample documentation indicating that endothelial dysfunction, which is a response to cardiovascular risk factors, typically precedes the development of atherosclerosis (54)