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Moreover, properly harnessing the Nrf2/HO-1 axis while managing its potential pro-ferroptotic risks may help maintain redox balance and curb iron overload, thereby offering an additional strategy to alleviate ferroptosis-driven damage in OA (202)

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Mitochondrial GSH preserves the mitochondrial integrity controlling mitochondrial ROS generation which promotes mitochondrial permeability transition pore opening, leading to mitochondrial dysfunction and cell death

(2017) RIP1-dependent reactive oxygen species production executes artesunate induced cell death in renal carcinoma Caki cells, Mol

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However, the same clinical trial revealed that only 6.2% of patients maintained any benefit at 6 months post-treatment compared to 0% in the placebo group