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We hypothesize that reduced CPT-I activity may contribute to persistent fatigue in these congenital hypothyroid patients, even after levothyroxine therapy, which alone may be insufficient to fully address the downstream metabolic consequences of TPO and TSHR gene mutations present in this cohort
This substantial weight loss is associated with favorable changes in metabolic parameters, including improvements in markers of insulin resistance (HOMA-IR), and beneficial shifts in lipid profiles
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