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Description

HFpEF exhibits more complexity and has more associated-comorbidities including hypertension compared to HFrEF, and systemic inflammation and its-induced metabolic injury are closely associated with the pathogenesis of HFpEF

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[] The brains main naturally occurring antioxidant is glutathione

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Unlike thermal (heat/cold) modalities, nsPFA directly impacts cellular structures while sparing noncellular tissue

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Under normal conditions, NRF2 is degraded by its negative regulator KEAP1 via the ubiquitin-proteasome pathway [10]

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