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This review starts by depicting the epidemiological features and pathogenic mechanisms of periodontitis, highlighting the vital mechanism of microbial community modefications, host immune responses, and genetic factors in disease development

Consistent with previous reports 16 , we observed mKate2-Annexin V in the distal fragment of the injured PLM of wildtype animals 1 h post injury, while in intact neurons mKate2 signal was detectable only in the cell body (Fig