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Porter C, Constantin-Teodosiu D, Constantin D, Leighton B, Poucher SM, Greenhaff PL

In summary, this section will elucidate the molecular mechanisms of ferroptosis in periodontitis, with a focus on the keystone pathogen P

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[42] demonstrated that allicin possessed hypolipidemic effects by upregulating low-density lipoprotein receptor (LDLR) expression through the activation of sterol regulatory element binding proteins 2 (SREBP2) and downregulating proprotein convertase subtilisin/kexin type 9 (PCSK9) expression via the suppression of hepatocyte nuclear factor 1 (HNF1), thereby enhancing the uptake of LDL by HepG2 cells

TXNIP promotes ferroptosis through NCOA4 mediated ferritinophagy

Methamphetamine-induced inhibition of mitochondrial complex II: roles of glutamate and peroxynitrite