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Whether IGF-1 LR3's prolonged receptor occupancy exacerbates this suppression more than native IGF-1 has not been studied directly, but the mechanism predicts a greater effect per unit time of receptor engagement [1, 18]

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[20] Population data from patients in clinical trials did indicate a reduction in sildenafil clearance when it was coadministered with CYP3A4 inhibitors

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