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It triggers a clean GH pulse with minimal effect on cortisol or prolactin

By performing acute A applications ex vivo , the authors proposed that soluble A might trigger the decrease of synaptic plasticity in neocortical pyramidal cell networks during early stages of AD pathogenesis by preferentially targeting postsynaptic AMPA receptors (Shemer et al., 2006)

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When the integrity of the BBB is breached, infiltrating blood components and immune cells activate perivascular glia, igniting an inflammatory cascade that accelerates neuronal damage