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While presumably such a shift would reduce mitochondrial production of reactive oxygen species, other sources including NADPH oxidase (NOX) (Wilkinson and Landreth, 2006

[86] showed that the reduction of GPx-1 activity in silenced human endothelial cells for GPx1 expression accelerated oxidative stress and increased nuclear factor kappa-light-chain-enhancer of activated B cells (NFB) and c-Jun-N-terminal kinase (JNK) activation

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Erratum in: JAMA 1988 Jun 10;259(22):3274

10.1089/ars.2011.4170 Antioxid