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They secrete a spectrum of profibrotic mediators, including TGF-1, IL-10, arginase-1, and CCL18, which promote fibroblast proliferation, myofibroblast differentiation, and excessive ECM deposition (109)

In preclinical studies, inhibition of AGE formation, or interventions that break their cross-linked structure, have been shown to delay the development of nephropathy, even without directly impacting blood sugar control (Forbes 2003)

This is particularly true for glutathione

Knockdown of RPL15 reduced cell proliferation and migration in vitro