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Mouse and rat models show that signals through proinflammatory receptors, such as the receptor for advanced glycation end products and Toll-like receptors, are suppressed by GLP-1 receptor agonists, possibly by central nervous system GLP-1 actions resulting in immunomodulation through parasympathetic and opioid efferent activity

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This does not mean, of course, that there is no need for caution, and clinical research is continuing to investigate these potential effects of GLP-1 use