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Specifically, retatrutide simultaneously activates three incretin and metabolic hormone receptors: GLP-1 (glucagon-like peptide-1) receptor promotes insulin secretion, reduces appetite, and slows gastric emptying [8] [9] GIP (glucose-dependent insulinotropic polypeptide) receptor enhances insulin release and may support fat metabolism [11] [12] Glucagon receptor preclinical and early clinical data suggest this may increase energy expenditure and promote hepatic fat breakdown, though glucagon receptor agonism can also raise blood glucose levels and the full implications in humans are still being characterised [5] [6] This triple mechanism of action is hypothesised to produce meaningful reductions in body weight by addressing multiple aspects of metabolic dysfunction

Muttenthaler, M., et al

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All of these receptors have a role in appetite, blood sugar, and energy balance

Abstract #1161911: acute renal failure: a rare and uncommon side effect of semaglutide