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Hsp90 also exerts a CaM-independent effect to promote eNOS activity (Takahashi and Mendelsohn, 2003)

A damaged skin barrier can perpetuate inflammation and oxidative stress, both of which trigger increased melanin production as a protective response

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Discussion Inflammation caused by degradation products of red blood cells (RBCs) following intracerebral hemorrhage leads to severe secondary brain injury, which is a major reason leading to neuronal apoptosis and worsening prognosis of ICH [24]