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Additionally, the two-way interactions and three-way interaction between time point, treatment, and baseline value was included in the model

The effect is subtle, progressive, and more stable than injection-based results

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Beyond the typical regulatory pathways mentioned above, ferroptosis is modulated by many regulators, such as p53, nuclear factor erythroid 2-related factor 2 (Nrf2) and AMP-activated protein kinase (AMPK)[37-39], among which p53 gets a lot of attention due to its controversial role in ferroptosis

Because aging also depletes the enzymes needed for natural glutathione synthesis, older individuals may require more sessions before observing visible anti-aging changes