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found that endothelial cell dysfunction, which is characterized by low nitric oxide (NO) bioavailability, greater reactive oxygen species production, the activation of abscission enzymes, and impaired extracellular lysosome-associated organelle function, triggers the degradation of the EG (57), leading to shear stress defects

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Animal data suggest subcutaneous routes required substantially higher doses than intravenous in rabbits, 1 mg/kg subcutaneous versus 25 mcg/kg IV, a 40-fold difference

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