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CSD has been demonstrated to activate the trigeminovascular system (TVS) both centrally and peripherally by exposing the dura to inflammatory molecules, indicating that susceptibility to CSD may contribute to the initiation of migraine attacks (26, 27)

This promising and attractive combination strategy is currently being evaluated in multiple clinical trials (NCT04343157, NCT04073966)

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This possibility is substantiated by the occurrence of hyperhomocysteinemia in humans exhibiting cystathionine -synthase deficiency [67, 68]

Interleukin-1 blockade with anakinra and heart failure following ST-segment elevation myocardial infarction: results from a pooled analysis of the VCUART clinical trials

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