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The influence of MTHFR on psychiatric diseases is mainly through reduction of MTHFR activity which results in elevation of homocysteine, reduction of DNA methylation-dependent methyl donor, finally induces hypomethylation, and then active disease-related genes

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The AITC treatment led to a dose-dependent growth inhibition of Col-0 WT seedlings as shown in Fig

Verdict: Limited evidence human endocrine studies are small or indirect, and DSIP has not been proven as an anxiety or cortisol-lowering therapy [810]