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Biochimica et Biophysica Acta (BBA) - General Subjects, 1860, 83643

In mouse models of cognitive dysfunction induced by streptozotocin, NAC inhibited the pathological increase in acetylcholinesterase (AChE) activity in the brain cortex and hippocampus, as well as butyrylcholinesterase (BChE) activity in the hippocampus [119]

Indeed, continuous exenatide exposure once weekly elicited a greater response than did short-acting exenatide, improving glycemic and lipids controls and lipoprotein metabolism, and decreasing systemic inflammation

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