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The complementation of the glr1 strain specifically with the cytosolic form of Glr1 resulted in wtlike OxD values in both the cytosol and the IMS, but not in the mitochondrial matrix (Figure 3B)

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In particular, different biological sources (human liver microsomes, liver S9 (microsomal and cytosolic) fractions, and human liver hepatocytes) were tested for their ability to form GSH-S conjugates with 10 commonly prescribed drugs [135]