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Suggested mechanisms include maintenance of the balance between Th1/Th2 and Th17/Treg cells, induction of M2 macrophage polarization, and anti-inflammatory and tissue repair effects (Du et al

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Previous studies have shown that independent and combined treatments with imidacloprid and esfenvalerate boosted caspase-3 activity in a dose-dependent manner (Ibrahim et al

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Alveolar surfactant in lungs of operated patients after acetylcysteine treatment