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499 Measurements of NA-induced changes in cyclic adenosine monophosphate (cAMP) revealed an increased sensitivity of this second messenger to changes in extracellular lactate, indicating altered signalling landscape at the plasma membrane in relation to lactate homoeostasis

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In early disease, the extent of AI varied widely from 5.4% (patient CAS-G) to 83.6% (SK-H) of the genome (Fig