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Endoplasmic reticulum stress induction and ERK1/2 activation contribute to nefazodone-induced toxicity in hepatic cells

Biological aging is the result of cellular decline now understood to be due to a combination of oxidative stress and telomere shortening

This provides humidity protection during the equilibration period

An interesting observation that deserves further exploration is the inherent ability of intra-portal insulin to regulate hepatic GH sensitivity that remains preserved despite prevailing changes in peripheral insulin resistance

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