10.1053/j.gastro.2022.01.023 25 LambC
Cellular mechanism of bilirubin It has been demonstrated that UCB is present at the cellular level in neurons, neuronal functions, and microscopic glia and that it causes neuronal death, demyelination, and gliosis
In the context of excess GPx-1, reduction of protein disulfides corresponded with diminished growth factor-mediated signaling and a reduction in mitochondrial function, characterized by a reduction in mitochondrial potential and a decrease in ATP generation (154)
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Thymosin Beta-4 (TB-500)-Ac-SDKP pathway: any relevance for the cardiovascular system
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