The pathogenesis of NAFLD, schematically represented in Figure 2, is multifactorial and multiple mechanisms have been proposed to explain the process of excessive liver lipid accumulation, with the subsequent possible inflammation and fibrosis (36)
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Research has demonstrated that these age-related functional declines can be partially reversed through NAD+ restoration, with studies showing that NAD+ precursor supplementation in aged animals can restore mitochondrial function, improve cognitive performance, and extend both healthspan and lifespan
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Cartilage destruction in RA is closely linked to the oxidative stress environment, and the hypoxic environment increases monocyte differentiation toward osteoclasts and elevates osteoclast activity (39) ( Figure 3 )
