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Eur J Pharmacol 2010

Although the pathogenesis of IH in patients with GHD treated with GH therapy is not fully understood, it is thought that GH increases IGF-1-mediated production of cerebral spinal fluid (CSF) from the choroidal plexus, leading to increased intracranial pressure [3]

Finally, membrane lipid composition and more importantly the long polyunsaturated fatty acid (PUFA) is playing a key role in ferroptosis sensitivity

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Normally, protective molecules such as those in the Bcl-2 family shield mitochondria from apoptosis induced by oxidation

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