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(PubMed) Packer L, Cadenas E

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However, a more recent study observed that physiological levels of oncogene (KRAS G12D and MYC) expression could trigger a NRF2-dependent antioxidant response to downregulate ROS levels in cells [8]

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Regarding pro-fibrogenic gene expression, the secretion of pro-collagen 1a1 was increased significantly (2-fold) as well, while no difference was found at the mRNA level except for ACTA2 , which was significantly increased (2-fold)