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A novel miR-338-3p/SLC1A5 axis has been identified as a key regulator of oxidative stress-induced ferroptosis in DR: high glucose significantly upregulates miR-338-3p expression in RPE cells, and miR-338-3p silencing reverses RPE cell death and ferroptosis ( Although studies have confirmed that the three components form a positive-feedback loop that drives DR progression and that antioxidant therapy can alleviate DR pathology, the key molecular triggers that initiate the loopsuch as the cell-specific mechanisms by which hyperglycemia induces excessive ROS production in different retinal cellsremain poorly defined

The antioxidant you've heard the most about is not the one your body relies on most

Storage requirements follow standard peptide protocols: refrigerated at 2-8 degrees Celsius after reconstitution

Perspectives on what we know, what we need to investigate and how to put knowledge into practice

Krhus ML, Knudsen E, Knop FK

2011;3:CD007749