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Genotype-specific differences in folate and homocysteine metabolism have implications for methylation-dependent regulatory pathways
Cysteine can usually be synthesized by the human body under normal physiological conditions if a sufficient quantity of methionine is available
High homocysteine could also mean you have an MTHFR gene mutation, which makes you less efficient at converting inactive folate to the active form of folate that breaks down homocysteine
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A., Sylvia, D., Nye, C
Telomeres are repetitive DNA sequences (TTAGGG/AATCCC) that cap the ends of chromosomes, protecting genetic material during cell division
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