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Mechanisms include direct DNA binding, affecting transcription factor activity, and epigenetic modifications

molecular weight 4,034 Da in the acylated form)

Dysfunction of ETC has been associated with various neurological diseases from infantile-onset neurodevelopmental regression (such as Leigh syndrome) to adult-onset optic neuropathy or peripheral neuropathy (such as Leber hereditary optic neuropathy (LHON) in cI deficiency, and neurogenic muscle weakness, ataxia, and retinitis pigmentosa (NARP) in cV deficiency) ( + (the metabolites of MPTP) which is a cI inhibitor (Nicklas et al., 1985

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