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DPP-4 inhibitors, the active comparator employed, are not biologically inert: by inhibiting dipeptidyl peptidase-4, they increase endogenous GLP-1 bioavailability, which may attenuate or confound the apparent advantage conferred by exogenous GLP-1 RA therapy

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Some cancer cells are very dependent on Mcl-1, which is the last line of defense keeping the cell from dying

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