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Bioinformatics analysis and in vitro and in vivo experiments have shown that the abnormal balance of thiol/disulfide bonds leading to oxidative stress is the pathological mechanism underlying neuronal damage in neurodegenerative diseases

The mRNA-LNP platforms lipid nanoparticle component used in preclinical vaccine studies is highly inflammatory

Dhar, H

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The molecular mechanisms could be the inhibition of the expression of apoptosis-related proteins BCL2-Associated X (BAX), Caspase-3 (CASP3), and poly ADP-ribose polymerase (PARP)