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In 2009, it was reported that TMAO degraded misfolded G98R mutant CRYAA by activating the ubiquitin-proteasome pathway, alleviating ERS and apoptosis of human lens epithelial B3 cells induced by aggregated G98R CrYAA

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While hot flashes are not listed as a recognized adverse effect in FDA prescribing information, some patients report experiencing these symptoms during treatment

Nitric oxide protects against ferroptosis by aborting the lipid peroxidation chain reaction

DM1: derivative of maytansine 1