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doi: 10.15625/2525-2518/19457 110

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This issue suggests that changes in energy expenditure due to altered thyroid function may be a primary factor leading to an increase in body weight, even with normal feedback regulation [59]

Interestingly, the thyroid profile went to normal when the glutathione drug administration was stopped

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In addition, ALOXs can directly or indirectly promote lipid oxidation in cell membranes to increase the sensitivity of cells to ferroptosis, especially under the catalysis of ferrous iron [34, 35]