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Typically, says Dr

Overall, the overexpression of AKR1C3 significantly enhances resistance to radiation in human PCa cells through the activation of the MAPK pathway

In the presence of oxygen, hydrated electrons, and H atoms react with molecular oxygen to produce radicals such as HO 2 , O 2 , apart from other aqueous free radicals.[] Enzymes such as glutathione peroxidase, catalase (CAT), and SOD protect mammalian cells from oxidative radiation damage.[] The sensitivity of the cells to ionizing radiation was increased in the presence of oxygen

A frequently cited reference point is the Witschi et al

Since the -2 subcluster is composed of cells exhibiting transcriptional signs of stress and the proportion of this subcluster was decreased upon liraglutide treatment, these data suggest that liraglutide-induced expression of XBP1 in the -1, -3, and -4 subclusters is a reflection of its protective action