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By enhancing perfusion, downregulating HIF-1, reversing chemoresistance, and modulating immunity, NO synergizes with radiotherapy, chemotherapy, and immunotherapy

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Activation of GLP-1R can inhibit apoptosis and inflammation by stimulating phosphoinositide 3-kinase (PI3K) and protein kinase A (PKA) [23]

Fraxetin alleviates BLM-Induced idiopathic pulmonary fibrosis by inhibiting NCOA4-mediated epithelial cell ferroptosis

For instance, central administration of GLP-1R agonists increases the ability of insulin to suppress endogenous glucose production (Knauf et al., 2005

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