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It was initially postulated to be a compensatory mechanism to the blood pressure lowering effect of GLP-1 RAs that was possibly mediated by their vasodilatory effect [9, 31] and/or increased urinary sodium excretion [32]

Most patients see measurable reduction in stubborn fat, particularly central and visceral fat, over a 6-12 week cycle

At the time of the study in early 2024, about 0.5% of the adult population in the US was using GLP - 1 therapies

Cells were lysed at 4 C in a radioimmunoprecipitation assay (RIPA) lysis buffer (Beyotime Biotechnology)

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