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Of particular note is a lack of neoplastic induction, since c-Met is recognized as an oncogene
Its native serin protease inhibitor (serpin) function is thought to play a role in controlling protease activities, such as those of kallikrein 7 (KLK7) 19,20,21,22
Although investigations have been undertaken looking at enzymes in isolation, little has been studied about how genetic differences act together to alter an individuals risk of paracetamol toxicity [16]
Steroid-refractory PD-(L)1 pneumonitis: incidence, clinical features, treatment, and outcomes
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GST non-enzymatic functions GSTs are also implicated as modulators of signal transduction pathways implicated in cell survival and apoptosis, where they control the activity of members of the mitogen-activated protein kinase (MAPK) family 24,25
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