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However, alcohol has direct pharmacological effects on neurons that differ from the effects of palatable food intake, which may explain why there are also examples of gut-brain peptides [e.g., neuropeptide Y (NPY)] that exhibit opposite effects on feeding and alcohol drinking (Thorsell et al., 2005a)

investigated the proximal signaling cascade and found that the mitochondrial phosphatase phosphoglycerate mutase family member 5 (PGAM5) is also involved in various pathways of regulated necrosis via interaction with another mitochondria-related protein dynamin-related protein 1 (Drp1) [213, 214]

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