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Activation by nitroreductase under hypoxia conditions, caused the release of SN38 with high selectivity and cytotoxicity toward biotin-positive tumor cells

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Exposing this model to -synuclein PFFs induces neuronal cell death, BBB permeability as well as altered expression of genes associated with autophagy, oxidative stress, mitochondrial dysfunction, and inflammation [202]

Thirdly, the observational design of this study, together with the absence of longitudinal molecular measurements, prevents definitive causal inference regarding selenium, brain aging, and the molecular candidates identified

Acta Physiologiae Plantarum 44, 112